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How to prevent and treat swine dysentery (Braxy-like dysentery)?

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Swine dysentery is an acute infectious disease caused by Treponema, clinically characterized by varying degrees of mucoid hemorrhagic diarrhea. The disease can occur in all four seasons, and pigs of all ages can be affected, but the incidence is high in piglets aged 2-3 months. The most acute form of this disease can cause sudden onset and death, which is also a sign that the pig herd is about to experience an outbreak. Acute symptoms then appear. At the beginning of the disease, the pigs are slightly dull, have reduced appetite, and their feces become hard with strip-like mucus on the surface. Diarrhea then develops rapidly. The feces are yellow, soft, or watery. In severe cases, within 1-2 days the feces

Swine dysentery is an acute infectious disease caused by Treponema, clinically characterized by varying degrees of mucoid hemorrhagic diarrhea. The disease can occur in all four seasons, and pigs of all ages can be affected, but the incidence is high in piglets aged 2-3 months. The most acute form of this disease can cause sudden onset and death, which is also a sign that the pig herd is about to experience an outbreak. Acute symptoms then appear. At the beginning of the disease, the pigs are slightly dull, have reduced appetite, and their feces become hard with strip-like mucus on the surface. Diarrhea then develops rapidly. The feces are yellow, soft, or watery. In severe cases, within 1-2 days the feces become filled with blood and mucus, or mixed with blood clots. Along with the appearance of diarrhea, the body temperature is slightly elevated (about 40.5°C), remains so for several days, and then drops to normal. Before death, the body temperature falls below normal. As the disease progresses, the affected pigs become depressed, lose weight, have increased thirst, and their feces become foul-smelling with increased blood, mucus, and fragments of necrotic epithelial tissue. The affected pigs gradually become emaciated, with pale skin, sunken abdomen, weakness in standing, extreme debility, and finally death.

The subacute form is milder, with diarrhea, more mucus and necrotic tissue fragments, less blood, and a longer disease course. There is progressive emaciation and stunted growth. Although the mortality rate is not high, growth and development are poor, feed conversion efficiency is reduced, and the impact on production is significant.

Necropsy reveals congestion and edema of the large intestine, especially the colon wall and mesentery, enlarged mesenteric lymph nodes, and the surface of the intestinal mucosa is often covered with gelatinous mucus and fibrinous exudate containing blood clots.

Prevention and treatment:

Mequindox, dimetridazole, and lincomycin, administered by intramuscular injection or orally, once a day, for 3-5 consecutive days as one course of treatment. Drugs should be used alternately to prevent the development of drug resistance.

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