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How to diagnose and treat chicken intestinal toxin syndrome? A detailed guide to practical methods

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1. Main manifestations of chicken intestinal toxin syndrome. Decreased feed intake, slowed weight gain or weight loss. Loose, unformed feces, with undigested feed in the droppings, color turning light yellow, yellowish-white, or fish-intestine-like droppings, carrot-like droppings. A marked increase in sudden deaths before dawn, or restlessness and agitation followed by limpness and death from exhaustion. Mostly occurs around 40 days of age. It is widespread in broiler flocks whether raised on floor litter or in cages, causing huge hidden economic losses, and is often overlooked by farmers and veterinary workers

1. Main manifestations of chicken intestinal toxin syndrome. Decreased feed intake, slowed weight gain or weight loss. Loose, unformed feces, with undigested feed in the droppings, color turning light yellow, yellowish-white, or fish-intestine-like droppings, carrot-like droppings. A marked increase in sudden deaths before dawn, or restlessness and agitation followed by limpness and death from exhaustion. Mostly occurs around 40 days of age.

In broiler flocks, it is widespread whether raised on floor litter or in cages, causing huge hidden economic losses, and is often overlooked by farmers and veterinary workers. Flocks with excessive density, excessive humidity, and poor ventilation have a higher incidence, more severe symptoms, and poorer treatment results. The higher the protein, energy, and vitamin content in the feed, the greater the chance of intestinal toxin syndrome occurring.

This disease can also occur in laying hen flocks. It mostly occurs just before or at the start of laying. Flocks suffering from intestinal toxin syndrome often also have respiratory syndrome. After intestinal toxin syndrome is cured, the symptoms of respiratory syndrome will also lessen or resolve on their own.

This disease is often misdiagnosed as general indigestion or small intestinal coccidiosis, but treatment with all anticoccidial drugs shows no obvious effect.

2. Main lesions of chicken intestinal toxin syndrome. Thickening of the intestinal mucosa, with the mucosa tending to slough off. There are hemorrhagic spots and necrotic foci on the intestinal mucosa. The intestinal wall becomes thinner, with relatively little intestinal content, containing poorly digested feed. The intestinal contents resemble fish intestines, carrots, or persimmons.

3. Causes of chicken intestinal toxin syndrome. Infection with small intestinal coccidia; massive proliferation of harmful bacteria in the intestine; changes in the intestinal environment; massive loss of electrolytes; the influence of protein, energy, and vitamins in the feed; excessive use of anticoccidial and antibacterial drugs; autointoxication.

4. Therapeutic effect of Puleweite. Each bag of Puleweite is diluted in 120 kg of water, with drinking water given for 12 hours per day. After 1 day of use, the feces become thicker and darker in color; after 2 days of use, feed intake increases markedly and weight gain speeds up; after 2 more days, the birds recover.

5. Situations in which Puleweite is not suitable. Diarrhea caused by moldy feed; diarrhea caused by excessive salt content in the feed; physiological diarrhea caused by increased water intake during hot, humid seasons; diarrhea caused by sudden heavy drinking after prolonged water deprivation

diarrhea caused by other infectious diseases.

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