How to effectively treat grass carp hepatobiliary syndrome

Grass carp hepatobiliary syndrome is a nutritional metabolic disorder disease occurring under high-density farming models that mainly rely on feeding pelleted compound feed, and it occurs mostly in summer. So, how to treat grass carp hepatobiliary syndrome?

1. How to treat grass carp hepatobiliary syndrome?

1. Cultivate good water quality and keep the water fresh;

2. Choose feed formulated by reputable professional feed manufacturers, feed scientifically, and raise reasonably;

3. Keep the bait fresh, prevent protein from spoiling and fat from oxidative decomposition, and prevent the feed from getting damp and moldy.

4. Do not add antibacterial drugs that harm fish livers to the feed at low doses for a long time; use drugs rationally and use fewer fish medicines with strong side effects and high residues.

5. During the farming process, patrol the pond frequently. If lesions appear in the fish liver, promptly add fresh water and feed medicated bait made with added vitamins and a small amount of rare elements or Chinese herbal medicines. Generally, feeding for 7 days is one course of treatment, which can effectively prevent the disease.

6. Once grass carp are found to have hepatobiliary syndrome, mix into the feed and feed liver-protecting and gallbladder-benefiting Chinese herbal medicines such as Sanhuang Powder and Banhuang Powder, or add VC and VE to the feed.

7. Reduce the amount of feed and feed some green grass or aquatic plants.

8. Use as few heavy metal ion insecticides such as copper sulfate as possible, to prevent excessive accumulation in the pond from damaging the liver.

2. Symptoms of grass carp hepatobiliary syndrome

1. As the temperature rises, fish begin to enter the peak growth season and have a strong appetite. But overfeeding makes it difficult for fish to digest and increases the burden on the liver and gallbladder. Feed with too high protein easily induces fat accumulation in the liver, damages liver function, and interferes with the normal physiological and biochemical metabolism of fish, while too high carbohydrate content causes disorders of sugar metabolism in fish, resulting in visceral fat accumulation and hindering normal function. The main site of the lesions is the liver. A large amount of liver glycogen accumulation and fat infiltration causes liver enlargement, lighter color, and a dull appearance. Severe fatty liver can also trigger liver lesions, causing the liver to lose normal function. Fish diseases characterized mainly by liver and gallbladder disorders are collectively called hepatobiliary syndrome.

2. When grass carp hepatobiliary syndrome is mild, the fish generally have no obvious symptoms, and there are no obvious changes in body color or body shape. Sometimes the edge of the tail fin turns white and dry, with only poor appetite and weak swimming; sometimes they are restless and anxious, and may even leap out of the water. When the condition is severe, the fish body color turns black, the color is dull, the body appears swollen, the scales are loose and fall off easily, swimming is irregular, they lose balance, or they remain motionless in the water; appetite decreases, reactions become sluggish, breathing is difficult, and they may even become comatose and turn over, dying soon after.

3. Dissection reveals that the liver color changes, appearing mottled, earth-yellow, yellow-brown, etc., and the gallbladder becomes enlarged and the bile turns black. In addition, the fish body has very poor stress resistance. When caught or transported, it often causes systemic congestion or bleeding in the fish body, and the fish die quickly after being taken out of the water, or die during transport.

After understanding grass carp hepatobiliary syndrome, we can know that this disease is mainly prevented, and once it is found, it needs to be treated promptly.